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1
Nucleotide excision repair-initiating proteins bind to oxidative DNA lesions in vivo.
Nucleotide excision repair-initiating proteins bind to oxidative DNA lesions in vivo.
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Nucleotide excision repair-initiating proteins bind to oxidative DNA lesions in vivo.

The Journal of cell biology, December 24, 2012, Vol.199(7), pp.1037-1046 [Peer Reviewed Journal]

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2
The core spliceosome as target and effector of non-canonical ATM signalling
The core spliceosome as target and effector of non-canonical ATM signalling
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The core spliceosome as target and effector of non-canonical ATM signalling

Nature, 2015 [Peer Reviewed Journal]

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3
Recognition of DNA damage by XPC coincides with disruption of the XPC-RAD23 complex.
Recognition of DNA damage by XPC coincides with disruption of the XPC-RAD23 complex.
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Recognition of DNA damage by XPC coincides with disruption of the XPC-RAD23 complex.

The Journal of cell biology, March 19, 2012, Vol.196(6), pp.681-688 [Peer Reviewed Journal]

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4
Translocation of Cockayne syndrome group A protein to the nuclear matrix: possible relevance to transcription-coupled DNA repair.
Translocation of Cockayne syndrome group A protein to the nuclear matrix: possible relevance to transcription-coupled DNA repair.
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Translocation of Cockayne syndrome group A protein to the nuclear matrix: possible relevance to transcription-coupled DNA repair.

Proceedings of the National Academy of Sciences of the United States of America, 2002, Vol.99(1), pp.201-206 [Peer Reviewed Journal]

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5
Targeted Apoptosis of Senescent Cells Restores Tissue Homeostasis in Response to Chemotoxicity and Aging
Targeted Apoptosis of Senescent Cells Restores Tissue Homeostasis in Response to Chemotoxicity and Aging
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Targeted Apoptosis of Senescent Cells Restores Tissue Homeostasis in Response to Chemotoxicity and Aging

Cell, 23 March 2017, Vol.169(1), pp.132-147.e16 [Peer Reviewed Journal]

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6
DNA damage sensitivity of SWI/SNF-deficient cells depends on TFIIH subunit p62/GTF2H1
DNA damage sensitivity of SWI/SNF-deficient cells depends on TFIIH subunit p62/GTF2H1
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DNA damage sensitivity of SWI/SNF-deficient cells depends on TFIIH subunit p62/GTF2H1

Nature Communications, 01 October 2018, Vol.9(1), pp.1-14 [Peer Reviewed Journal]

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7
A new progeroid syndrome reveals that genotoxic stress suppresses the somatotroph axis
A new progeroid syndrome reveals that genotoxic stress suppresses the somatotroph axis
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A new progeroid syndrome reveals that genotoxic stress suppresses the somatotroph axis

Nature, 2006, Vol.444(7122), p.1038 [Peer Reviewed Journal]

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8
Different effects of CSA and CSB deficiency on sensitivity to oxidative DNA damage.
Different effects of CSA and CSB deficiency on sensitivity to oxidative DNA damage.
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Different effects of CSA and CSB deficiency on sensitivity to oxidative DNA damage.

Molecular and cellular biology, September 2004, Vol.24(18), pp.7941-7948 [Peer Reviewed Journal]

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A 3' --> 5' XPB helicase defect in repair/transcription factor TFIIH of xeroderma pigmentosum group B affects both DNA repair and transcription.
A 3' --> 5' XPB helicase defect in repair/transcription factor TFIIH of xeroderma pigmentosum group B affects both DNA repair and transcription.
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A 3' --> 5' XPB helicase defect in repair/transcription factor TFIIH of xeroderma pigmentosum group B affects both DNA repair and transcription.

Journal of Biological Chemistry, 1996, Vol.271(27), pp.15898-15904 [Peer Reviewed Journal]

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10
The second zinc-finger domain of poly(ADP-ribose) polymerase determines specificity for single-stranded breaks in DNA.
The second zinc-finger domain of poly(ADP-ribose) polymerase determines specificity for single-stranded breaks in DNA.
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The second zinc-finger domain of poly(ADP-ribose) polymerase determines specificity for single-stranded breaks in DNA.

Proceedings of the National Academy of Sciences of the United States of America, 1990, Vol.87, pp.2290-2294 [Peer Reviewed Journal]

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by this Author/Contributor:

  1. Hoeijmakers, Jan
  2. Department of Molecular Genetics
  3. Erasmus MC: University Medical Center Rotterdam
  4. Hoeijmakers, J.H.J.
  5. Hoeijmakers, Jhj

on this subject:

  1. DNA Repair
  2. Animals
  3. Human
  4. Biology
  5. Humans

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